Zhonghua Nan Ke Xue. 2008 Jul;14(7):602-5.
[Tshooj hauv Suav]
Wang T1, Liu B, Wu ZJ, Yang B, Liu JH, Wang JK, Wang SG, Yang WM, Nej ZQ.
Abstract
HAIS TXOG:
Yuav kawm txog cov kev hloov ntawm hypothalamus metabolism hauv cov neeg mob uas muaj kev puas hlwb erectile kawg (ED) thiaj li tau txais qee qhov qhia txog cov kab mob tau pathogenic thiab pathophysiological mechanism ntawm qhov teeb meem.
KEV HLOOV:
Rau rau cov neeg mob hlwb ED thiab 4 cov neeg tuaj yeem pab dawb tau kawm los ntawm positron emission tomography (Tsom) rau cov yam ntxwv ntawm hypothalamus qabzib metabolism. Ua raws li cov stimulation deev txiv hmab txiv ntoo, cov concentration ntawm fluorine-18-fluorodeoxyglucose (18 F-FDG) tau txiav txim siab thiab qhov sib piv ntawm sab laug (txoj) hypothalamus suav rau lub paj hlwb count.
COV LUS QHIA:
Audiovisual deev kev sib raug zoo ntau ntxiv 18F-FDG hauv cov neeg tuaj pab dawb (sab laug: 1.026 +/- 0.115 vs 2.400 +/- 0.210; txoj cai: 1.003 +/- 0.187 vs 2.389 +0.196, P <0.05) piv rau cov neeg mob hlwb (EDogenic ED) ( sab laug: 2.781 +/- 0.156 vs 2.769 +/- 0.223; txoj cai: 2.809 +/- 0.129 vs 2.793 +/- 0.217, P> 0.05).
XAUS:
Psychogenic ED tej zaum yuav tsis yooj yim rau cov kab mob hauv lub cev; tus hypothalamus tuaj yeem koom tes rau hauv txoj kev mob ntsws ntawm qhov teeb meem.
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