Sinapsis. 2004 Jul;53(1):20-7.
Bradley fcilicet KC1, Series Mullins, RL Meisel, VJ Cicero.
Abstract
Medicamenta in abusu producendum longa-term mutationes neurotransmission et dopamine in intracellular signalling receptor est. Similes mutationes sunt in neuronal operatio mediante motivated partum. His underlying neuroadaptations his mechanisms ut explorarent cellular sexualem experientia, congeriem haec Insecta titillatione D1 dopamine receptores, G, proteins, et comparari Galectin erat autem fornicatur in nuclei caudati accumbens et hamsters simplices et in his peritus feminam sexualem moribus. Immediate excitare in Galectin cum forskolin vel indirecte activation de G-proteins in GPP (NH) p produci pondere-dependens crescit in constitutione rerum Insecta in in nucleus accumbens et callosum, cum nullus effectus sexualem experientia ad mensuras superiores. Auctis specificae D1 receptor GPP excitanda (NH) p nucleus inducatur Cholecystokinin accumbens industriae ac animalium caudati. Interestingly, hoc decorarat atque auxerat, excitanda erat adhuc in accumbens membranarum modo a vivente nucleum creaverunt, sed non ex callosum, qui fornicatur periti hamsters comparari responsionis innocentium feminas gigni. Haec consequitur et demonstrat in can expertus per turpitudinem properabant sensitize mesolimbic dopamine et tractuum sensitisation occurs in hoc augeatur D1 receptor-mediata signaling.
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